About the NpC ("Neuroplex-C") Project

This is a brief summary. Go to the NpC website and learn more!

Chronic, sustained stressor agents – molecular-scale but originating from environment by intake and by consequences of psychosocial behavioral processes – contribute to deterioration of intracellular communication pathways including energy transfer within microtubulin and axonal sheaths. Consequences include signal turbulence and interference within the autonomic nervous system and also involving the peripheral nervous system and muscular-skeletal network. Sustained patterns of dissonance and arrthythmia including spiral, chaotic and knot/vortex wave interference arise within principally cardiovascular, pulmonary, and gastrointestinal neuromuscular action, giving rise to several forms of long-term syndromic disease.

At the fundamental molecular scale this involves cellular structures including microtubulin, actin and other long-filament polymer chains and the disruption of normal, efficient energy transfer. Genetic factors are indicated in several conditions as contributing and exacerbating factors if not fundamental causes.

Inflammation effects including from infectious disease (e.g., COVID-19) can progress into and exacerbate pre-existing instabilities, leading to rapid and/or long-term deterioration into several autoimmune and syndromic diseases.